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Given the high expression of ABCG2 in the intestine, it may primarily facilitate intestinal urate excretion rather than renal excretion (Figure 2) [49]

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Under homeostatic conditions, transient activation of NRF2 is cytoprotective, allowing for adaptation to various cell intrinsic (i.e., reactive oxygen species (ROS) generated by mitochondrial function) and extrinsic (i.e., xenobiotic) stressors (Kensler et al, 2007

Finally, dietary glutamate prevented mycotoxin-induced impairments in intestinal barrier function and morphology in young piglets, suggesting that free glutamate may also play a role in the prevention of intestinal damage (135)